Hi, my modem is going on the blink! So I will post this info I found, QUICK. Most of what was in my former post were QUOTES, lol. I thought you knew!
I haven't even read all of the below information. If any is scary, don't believe it. I was constantly being knocked off the net, while I was searching for info.
If a computer analysis is what gave you all of these results, do NOT worry. They have give readings on me that scared the heck out of me, while in the ER and hospital. The cardio always explained that the computer was wrong. He would take his time and try to show me why. I have been in and out of hospitals for 3 years, so trust me.
Also the equipment can be out of calibration, the tech can misplace the electrodes. I don't like anything except for a 12, I say again, 12 lead EKG. If they truly find something mysterious, they will run more EKGs on you. Cheers :)
Leads I, F, and V5-V6 are called lateral leads. Abnormality in these leads indicates pathology on the lateral, upper surface of the heart.
T Wave Abnormalities
T wave abnormalities can provide added evidence to support clinical diagnosis. Except for hyperkalemia, T wave abnormality alone is not diagnostic of any particular condition. The T wave must be considered along with QRS and ST segment abnormalities. T waves will usually be abnormal in ventricular hypertrophy, left bundle branch block, chronic pericarditis, and in electrolyte abnormality.
Tall, peaked T waves occur due to hyperkalemia. If the tall T waves are seen throughout the ECG, general hyperkalemia is present. P waves will be small, PR interval short.
When typical tall, peaked T waves are seen only within a specific set of cardiac leads, it suggests impending infarction. The tall Ts are due to potassium leak through damaged membranes in the area of the infarct.
T Wave Categories
Tall, peaked = hyperkalemia if generalized
infarction if localized
Inverted = evolving infarction
acute cerebral disease
other cardiac disease
Flattened = nonspecific
In chronic pericarditis, T waves show wide-spread inversion, not corresponding to any coronary artery distribution. General inversion of T waves can also be due to an evolving global subendocardial infarct.
Inverted T waves are seen during the evolution of myocardial infarction. The T inversion appears in the leads "looking at" the infarcted area. Several hours after an infarct, T waves begin to invert. T wave inversion may persist for months.
Left ventricular hypertrophy or strain commonly causes T wave inversion. In "strain" pattern, the ST segment slopes down to an inverted T in the leads "looking at" the affected ventricle.
Right ventricular hypertrophy or acute ventricular strain can produce changes in the right precordial leads, V1 and V2. The T wave will be inverted over right heart leads showing evidence of hypertrophy and strain.
Left bundle branch block can cause ST depression and inverted T waves in leads I, L, and V5-V6. The ST depression is usually not great. The T wave tends to be oriented opposite the QRS in LBBB.
Flat T waves can be seen in many conditions, including ischemia, cardiac scar, evolving infarction, and electrolyte abnormality (such as hypokalemia).
In acute cerebral disease, such as intracranial hemorrhage, elongated or bizzare T waves may be seen. These Ts are often biphasic or deeply and sharply inverted. The QT interval is often dramatically lengthened (0.5 to 0.7 seconds).