IBS does not have a fungal etiology.

 

 

Many people who go on low carb and low fat diets may have their IBS symptoms helped. One by reducing gas from bacteria, second carbs breakdown to Tryptophan an amino acid, which then helps create serotonin. Serotonin is involved in IBS, there has been extensive research on the subject.

 

Gut bacteria and yeast are two different issues. There are some 500 gut bacteria in the gi tract.

 

 

 

"Four infections are classified in the superficial mycoses. Black piedra, caused by Piedraia hortai, and white piedra, caused by Trichos**** beigleii, are infections of the hair. The skin infections include tinea nigra, caused by Exophiala werneckii, and tinea versicolor, caused by M. furfur. Where the skin is involved, the infections are limited to the outermost layers of the stratum corneum; in the case of hairs, the infection is limited to the cuticle. In general, these infections cause no physical discomfort to the patient, and the disease is brought to the attention of the physician for cosmetic reasons."

http://www.answers.com/topic/fungal-infection

" There is increasing evidence for yeasts being able to cause IBS-symptoms"

This statement is outdated and there are specific IBS symptoms yeast doesn't cause. Lots of things can cause D for example or some digestive symptoms. IBS symptoms are a specific cluster of symptoms.

Foods don't cause IBS.

Yeast infections also cause inflammation. Macroscopic inflammation has been seen in some IBSers, under an electron microscope, but it was not due to yeast. With an electron microscope you can observe individual cells.  Millions of IBSers have had colonoscopies and no overt inflammation of the colon or yeast overgrowth has been found. It would be visable.

again carbs breakdown to serotonin and that IS a probelms in IBS. Sugar can cause an increase in bacteria and more gas as they are broken down in the digestive tract and its the pressure the gas puts on the hypersensitive bowel by distension that can cause symptoms and pain.

While I am glad to hear your personally symptoms free, it doesn't mean yeast is a "cause" of IBS.  There is also a ton of research going on right now in regards to gut bacteria and IBS.

 

http://www.iffgd.org/store/viewproduct/222

 

Video Corner: Gut Flora, Probiotics and Antibiotics

http://www.aboutibs.org/site/learning-center/video-corner/gutflora

This was a stduy of gut microflora and the only thing they found was that "Bifidobacteria" were low.

http://www.ncbi.nlm.nih.gov/pubmed/19533811?itool=EntrezSystem2.PEntrez.Pubmed.Pubmed_ResultsPanel.Pubmed_RVDocSum&ordinalpos=10

Also many people can have inflammation in the gi tract and NO pain. Pain or discomfort are a must for an IBS diagnoses.

There is a ton of research being down in gut microflora and its influence on digestion on each of the 500 gut micro-organisims. It is not the only research being done and there is a lot more they have found dysfuntioning.

 

In recent research a small percentage of IBSers have been found to also have another condition called SIBO as well as IBS. So a person can have more then one condition.

An organic GI yeast infection is NOT a diagnoses of IBS. It can also be life threatening.

also

http://www.doctorfungus.org/mycoses/human/candida/Chronic_Candidiasis.htm

 

 

 

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I am not a doctor. All information I present is for educational purposes only and should not be subsituted for the advise of a qualified health care provider.

Please make sure you have your symptoms diagnosed by a medical practitioner or a doctor.

 

Even if the involvement of yeasts in the aetiology of IBS still remains unclear, there is increasing evidence for yeasts being able to cause IBS-symptoms in sensitized patients via Candida products, antigens and cross-antigens. But more research is needed before antifungal treatment can be recommended as a first line treatment for IBS.

 

Yeast infections usually remain localized upon a moist surface, but in prolonged cases the yeast can shift to a fungal form which sinks root-like rhizoids under the surface of the mucosa. This usually happens invisibly within the intestinal tract, and when it does, toxins and other substances which are normally prevented from penetrating the surface of the intestinal lining are given a route to invade the rest of the body, causing “leaky gut syndrome”.

I'm still a firm believer my symptoms are/were fungus related. With over 400 kinds of fungi harmful to man, it may not only be "candida" that could be a problem for those of us with health issues.

 

IBS is not caused by candida or fungus.

 

They know some foods trigger IBS and has nothing to do with fungus, but for other chemical reasons.

 

The second study you posted is from 2005 and there has been an explosion in IBS research since then your leaving out of the picture here. There are almost no candida and IBS references in Pubmed out of 19 million IBS references.

 

In 20 years of so called "candida syndrome" it has never been found in ANYONE.

 

"invisibly within the intestinal tract," NOT WITH AN ELECTRON MICROSCOPE it isn't.

 

Three mechansims contribute to IBS.

MOTILITY
In healthy subjects, stress can increase motility in the esophagus, stomach, small and large
intestine and colon. Abnormal motility can generate a variety of GI symptoms including
vomiting, diarrhea, constipation, acute abdominal pain, and fecal incontinence. Functional GI
patients have even greater increased motility in response to stressors in comparison to normal
subjects. While abnormal motility plays a vital role in understanding many of the functional GI
disorders and their symptoms, it is not sufficient to explain reports of chronic or recurrent
abdominal pain.

VISCERAL HYPERSENSITIVITYVisceral hypersensitivity helps to account for disorders associated with chronic or recurrent pain, which are not well correlated with changes in gastrointestinal motility, and in some cases, where motility disturbances do not exist. Patients suffering from visceral hypersensitivity have a lower pain threshold with balloon distension of the bowel or have increased sensitivity to even normal intestinal function. Additionally, there may be an increased or unusual area of somatic referral of visceral pain. Recently it has been concluded that visceral hypersensitivity may be induced in response to rectal or colonic distension in normal subjects, and to a greater degree, in persons with IBS. Therefore, it is possible that the pain of functional GI disorders may relate to
sensitization resulting from chronic abnormal motor hyperactivity, GI infection, or trauma/injury
to the viscera. http://www.med.unc.edu/ibsThe UNC Center for Functional GI& Motility Disorders5

BRAIN-GUT AXIS
The concept of brain-gut interactions brings together observations relating to motility and visceral hypersensitivity and their modulation by psychosocial factors. By integrating intestinal and CNS central nervous system activity, the brain-gut axis explains the symptoms relating to functional GI disorders. In other words, senses such as vision and smell, as well as enteroceptive
information (i.e. emotion and thought) have the capability to affect gastrointestinal sensation,
motility, secretion, and inflammation. Conversely, viscerotopic effects reciprocally affect central
pain perception, mood, and behavior. For example, spontaneously induced contractions of the
colon in rats leads to activation of the locus coeruleus in the pons, an area closely connected to
pain and emotional centers in the brain. Jointly, the increased arousal or anxiety is associated
with a decrease in the frequency of MMC activity of the small bowel possibly mediated by stress
hormones in the brain. Based on these observations, it is no longer rational to try to discriminate
whether physiological or psychological factors produce pain or other bowel symptoms. Instead,
the Functional GI disorders are understood in terms of dysregulation of brain-gut function, and
the task is to determine to what degree each is remediable. Therefore, a treatment approach
consistent with the concept of brain-gut dysfunction may focus on the neuropeptides and
receptors that are present in both enteric and central nervous systems. "

http://216.109.125.130/search/cache?ei=UTF...=1&.intl=us

 

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I am not a doctor. All information I present is for educational purposes only and should not be subsituted for the advise of a qualified health care provider.

Please make sure you have your symptoms diagnosed by a medical practitioner or a doctor.

 

My bet is still on fungus having a part ( and not necessarily "ALL") in gastrointestinal health issues....especially when mycotoxins can be introduced into our body through foods such as grains and cereals, or from taking antibiotics. How many of us can say we have never been on antibiotics, or eaten grains.