Alot of men have various degrees of urinary tract infections UTI's or normal enlargement of the prostate gland as we age BPH and other causes of restriction to urinary flow. Here is an interesting link with some particular trades that have men more at risk for bladder related cancer and UTI's. This link also discusses some of the worries or risks of having total urinary restriction which can even lead to death scenarios. So, if you are one having very significant urinary changes it would be wise to have them investigated. Myself waited way to long, denial biases or whatever, had urinary changes and issues of stream and going...waited it out and wondered if it would go away (not). Ended up with total urinary blockage back in 2002, and into emergency room...the pain is overwhelming...good thing I didn't wait much longer.
No data are available on incidence and prevalence of urinary obstruction in unselected populations. Most epidemiologic studies of obstruction are in selected populations or autopsy studies. In large surveys of elderly men for symptoms of urinary obstruction, a prevalence of 20-35% has been estimated. Most (60%) of the men surveyed with moderately severe to severe symptoms of prostatism did not consult their physicians with these symptoms. Postmortem examinations have found hydronephrosis in 3.8% of adults and 2.0% of children.
Urinary tract obstruction may lead to acute or chronic renal insufficiency or overt kidney failure. Obstruction may lead to a salt-losing nephropathy and urinary concentrating defects. Renal tubular acidosis (RTA) type IV, hyperkalemia, hypomagnesia, and hypophosphatemia are common sequelae of chronic obstruction. Although acute or chronic obstruction may cause urinary tract infection (UTI), other sequelae such as renal calculi, hypertension, and polycythemia are associated with a chronic setting. Ascites is a common sequela of neonatal obstruction syndrome. In cases of acute obstruction, a postobstructive diuresis following relief of the problem is well described...
In cases of both acute and chronic obstructive uropathy, occupational exposure history may be beneficial. For example, in textile manufactures, shipyard workers, roofers, or asbestos miners, retroperitoneal fibrosis due to asbestos-induced mesothelioma should be considered. Bladder cancer–induced outlet obstruction may occur in textile workers, rubber manufacturing workers, leather workers, painters, hairdressers, or drill press workers exposed to alpha- or beta-naphthylamine, 4-aminobiphenyl, benzidine, chlornaphazine, 4-chlor-o-toluidine, 2-chloroaniline, phenacetin compounds, benzidine azo dyes, or methylenedianiline.
- Signs of dehydration and intravascular volume depletion can be seen as a result of urinary concentrating defects associated with partial obstruction. Peripheral edema, hypertension, and signs of congestive heart failure from fluid overload may be observed in obstruction from renal failure.
- Palpable kidney or bladder provides direct evidence of a dilated urinary collection system.
Link- http://emedicine.medscape.com/article/778456-overview (for full article)
Message is find out what your urinary issues are about and try to handle it sooner than later. Maybe someone soon will have this message talk to them directly. I only need Flomax for intial huge improvement, but underlying it all was a huge case of PCa as the real cause in my situation.
Dx-2002 total urinary blockage, bPsa 46.6 12/12 biopsies all loaded 75-95% vol.; Gleasons scores 7,8,9's (2-sets), gland size 35, ct and bone scans look clear- ADT3 5 months prior to radiations neutron/photon 2-machines, cont'd. ADT3, quit after 2 yrs. switched to DES 1-mg, off 1+ yr., controlled well, resumed, used intermittently, resumed useage
Post Edited (zufus) : 9/25/2010 8:47:31 AM (GMT-6)