This diet is supposed to correct the flora and allow improved processing of fiber resulting in higher butyrate and healing mucosal layers. Anybody try it ? Been on it for a few days now. Navy beans over Black beans is how I am going to proceed. Less ordinary fiber that way.www.ncbi.nlm.nih.gov/pubmed/10647628
Dietary resistant starch and chronic inflammatory bowel diseases.
These studies were performed to test the benefit of resistant starch on ulcerative colitis via prebiotic and butyrate effects. Butyrate, propionate, and acetate are produced in the colon of mammals as a result of microbial fermentation of resistant starch and other dietary fibers. Butyrate plays an important role in the colonic mucosal growth and epithelial proliferation. A reduction in the colonic butyrate level induces chronic mucosal atrophy. Short-chain fatty acid enemas increase mucosal generation, crypt length, and DNA content of the colonocytes. They also ameliorate symptoms of ulcerative colitis in human patients and rats injected with trinitrobenzene sulfonic acid (TNBS). Butyrate, and also to a lesser degree propionate, are substrates for the aerobic energy metabolism, and trophic factors of the colonocytes. Adverse butyrate effects occur in normal and neoplastic colonic cells. In normal cells, butyrate induces proliferation at the crypt base, while inhibiting proliferation at the crypt surface. In neoplastic cells, butyrate inhibits DNA synthesis and arrests cell growth in the G1 phase of the cell cycle. The improvement of the TNBS-induced colonic inflammation occurred earlier in the resistant starch (RS)-fed rats than in the RS-free group. This benefit coincided with activation of colonic epithelial cell proliferation and the subsequent restoration of apoptosis. The noncollagenous basement membrane protein laminin was regenerated initially in the RS-fed group, demonstrating what could be a considered lower damage to the intestinal barrier function. The calculation of intestinal short-chain fatty acid absorption confirmed this conclusion. The uptake of short-chain fatty acids in the colon is strongly inhibited in the RS-free group, but only slightly reduced in the animals fed with RS. Additionally, RS enhanced the growth of intestinal bacteria assumed to promote health. Further studies involving patients suffering from ulcerative colitis are necessary to determine the importance of RS in the therapy of a number of intestinal diseases and the maintenance of health.en.wikipedia.org/wiki/Resistant_starch
Foods naturally rich in resistant starch may be beneficial in individuals with ulcerative colitis. One study presented at the 7th Congress of European Crohn’s and Colitis Organization (ECCO) meeting, Feb 16-18, 2012 in Barcelona, Spain found that resistant starch combined with wheat bran effectively treated ulcerative colitis./www.ecco-ibd.eu/publications/congress-abstract-s/item/p208-abnorm.html
Abnormal fibre utilisation and gut transit in ulcerative colitis in remission: A potential new target for dietary intervention
Conclusions: Fibre utilisation in the gut of patients with UC in remission is impaired. High WB/RS supplementation enhanced fibre utilisation and normalised WGTT. As it is well tolerated, its corrective effect on fermentation and transit may translate into therapeutic benefits during remission in patients with UC.