Huge article and of course I did not read it all but some here might read through it and might have already posted on it such as oldMike or DBwithUC.aem.asm.org/content/78/18/6656.full
What I found interesting was the paragraph referencing source 44 that intersected RS and Elafin :
Linked Article said...
Microbiota changes induced by feeding RS also increased the expression of a gene encoding a serine protease, Ela1. Serine proteases have an important role in the induction of inflammation at mucosal surfaces (16, 22) and defense against infection (13). Inhibition of serine protease has been shown to reduce inflammation and tissue damage (11, 14, 44). However, serine proteases are also able to promote the generation of immune-suppressing CD4+ FOXP3+ regulatory T cells in vitro (34). The contrasting stimulatory and suppressive effects of serine proteases on inflammation suggest they may play a role in feedback mechanisms that prevent excessive inflammation caused by bacterial stimulation. In addition to changes in gene transcription, RS-induced changes in the microbiota also increased the abundance of mucus-producing goblet cells, another potential defensive response to the altered microbial makeup. Our results strongly suggest that alterations in microbiota composition impart stimuli to the host that result in physiological responses that attempt to maintain homeostasis.
More research is required to reveal the bacterial physiological attributes of the blooms in relation to RS, particularly in light of the broad changes in microbiota composition and host physiology seen by feeding even small doses of RS at an early age.