Post Linx manometry

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Regular Member

Date Joined Oct 2012
Total Posts : 322
   Posted 1/1/2013 5:52 PM (GMT -6)   
Does post-LINX manometry confirm increased LES pressures?

I am having trouble finding relevant data both on this forum and the Linx papers.

Anyone has any info that you could share ?

Forum Moderator

Date Joined Feb 2009
Total Posts : 7180
   Posted 1/2/2013 9:05 AM (GMT -6)   
Hi gerd_hater,
I'm no expert, but I have had a manometry post-Nissen, and I believe that it was able to determine how well the wrap was holding.  While it's not a LINX, they're both supposed to do the same thing, so it makes sense that it could determine whether or not it was offering the proper resistance. 
I do remember them mentioning that the swallowing reflex the base of the esophagus was "abnormal" due to the resistance of the wrap, but this is consistent with having the surgery and is actually a good sign that there is, indeed, a barrier there.
Good luck, and Happy New Year!
GERD/Heartburn Moderator
Nissen Fundoplication 2/09

"Whatever you fight, you strengthen, and what you resist, persists.”

“Worry pretends to be necessary but serves no useful purpose”

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Regular Member

Date Joined Oct 2012
Total Posts : 322
   Posted 1/2/2013 7:29 PM (GMT -6)   
Hi Denise, Thanks for the info. Did not know the abnormal swallowing reflex post Nissen.


Pat Tall
Veteran Member

Date Joined Aug 2012
Total Posts : 950
   Posted 1/2/2013 8:18 PM (GMT -6)   
GH. Yes, have seen manometry tests for pre and post Linx implant. The post readings are looked at to see if the Linx is too tight. The reading also reflects the effectiveness of the Linx to support the LES. This also tells the docs if dilatation of LES would be needed.

Regular Member

Date Joined Oct 2012
Total Posts : 322
   Posted 1/4/2013 12:14 AM (GMT -6)   
Found the LES post LINX from the original paper

Thirty-two patients had both baseline and one-year postoperative manometric testing available for comparison. LES
resting pressure increased from 6.5 mmHg to 14.6 mmHg
(p<.005) in the 9 patients with a hypotensive (less than 10
mmHg) LES pressure. In the 23 patients with normal LES
pressure at baseline, no significant changes in pressure
occurred (baseline=18.1 mmHg, postoperative=19.7 mmHg).
There were no statistically significant changes in the overall
or abdominal length of the LES. Similarly, there were no
statistically significant changes in the amplitude of esophageal

For comparison with Endostim,
All patients were successfully implanted nine subjects received high frequency, low energy, and four subjects received low frequency, high energy stimulation. Both types of stimulation significantly increased resting LESP: from 8.6 mmHg (95% CI 4.1–13.1) to 16.6 mmHg (95% CI 10.8–19.2), P < 0.001 with low energy stimulation and from 9.2 mmHg (95% CI 2.0–16.3) to 16.5 mmHg (95% CI 2.7–30.1), P = 0.03 with high energy stimulation.
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